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Monday, August 10, 2020 | History

2 edition of Drug and hormone resistance in neoplasia found in the catalog.

Drug and hormone resistance in neoplasia

Drug and hormone resistance in neoplasia

  • 379 Want to read
  • 10 Currently reading

Published by CRC Press in Boca Raton, Fla .
Written in English

    Subjects:
  • Drug resistance in cancer cells.,
  • Cancer -- Chemotherapy -- Complications.,
  • Hormone therapy -- Complications.,
  • Cancer -- Endocrine aspects.,
  • Hormone resistance.,
  • Drug resistance.,
  • Hormones -- Pharmacodynamics.,
  • Neoplasms -- Drug therapy.

  • Edition Notes

    Includes bibliographical references and index.

    Statementeditors, Nicholas Bruchovsky, James H. Goldie.
    ContributionsBruchovsky, Nicholas., Goldie, James H.
    Classifications
    LC ClassificationsRC271.C5 D77 1982
    The Physical Object
    Pagination2 v. :
    ID Numbers
    Open LibraryOL3485018M
    ISBN 100849365163, 0849365171
    LC Control Number82004530

    Multiple endocrine neoplasia type 1 (MEN1) is an inherited disorder that causes hormone-secreting tumors in the duodenum and the endocrine glands-most often the parathyroid, pancreas, and pituitary. Overactive parathyroid glands can lead to tiredness, weakness, muscle or bone pain, constipation, indigestion, kidney stones, or thinning of bones. Papillary thyroid carcinoma: Most common (75%), Orphan-Annie nuclei, psammoma bodies. Follicular thyroid carcinoma: Medullary thyroid carcinoma: Neoplastic parafollicular cells (C-cells), ↑calcitonin, derived embryologically from ultimobranchial body, associated with mutations in RET gene (same as in MEN type 2). Anaplastic thyroid carcinoma: Rare, poor prognosis.

    Follow the links to read common uses, side effects, dosage details and read user reviews for the drugs listed below. 62 medications found for multiple endocrine neoplasia Sorted . In the late s, treatment of hormone receptor-positive breast cancer was revolutionized by the introduction of the drug tamoxifen. Since then, a multitude of successful new hormone-based (endocrine) drugs have followed. But despite this formidable arsenal, not all patients respond to these drugs, and many who do respond eventually relapse.

      Recent Progress in Hormone Research, Volume 46 provides a superior summary of the developments in the field of hormone research. The book discusses the molecular basis of androgen insensitivity; the tissue-specific expression of the growth hormone gene and its control by growth hormone factor-1; and the molecular characterization of mammalian tachykinin Brand: Elsevier Science. The major forms of multiple endocrine neoplasia are called type 1, and type 2. These types are distinguised by the genes involved, the types of hormones made, and the characteristic signs and symptoms. MEN Type 1 is the most common and affects approximately 1 in 30, and is caused by a heritable defect in the MEN-1 tumour suppressor gene.


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Drug and hormone resistance in neoplasia Download PDF EPUB FB2

Drug and Hormone Resistance in Neoplasia: Clinical Concepts: Medicine & Health Science Books @ ISBN: OCLC Number: Description: 2 volumes: illustrations ; 27 cm: Contents: v. Basic concepts --v The book describes the colon cancer as a model for resistance to antineoplastic drugs; mathematical modeling of drug resistance; and the mechanism of induced gene amplification in mammalian cells.

The text also discusses the cellular concomitants of multidrug resistance; resistance to alkylating agents; and the phosphoprotein and protein kinase C changes in human multidrug-resistant cancer. This chapter discusses the characteristic features, epidemiology, pathophysiology, clinical features, complications, and management of endocrine-related genetic disorders and diseases such as McCune–Albright syndrome, endocrine tumours in neurofibromatosis type 1, Von Hippel–Lindau disease (VHL), Carney complex, Cowden syndrome, and multiple endocrine neoplasia Author: Helen E.

Turner. In: Bruchovsky N, Goldie JH (eds) Drug and hormone resistance in neoplasia, vol I. CRC, Boca Raton, pp – Google Scholar Isaacs JT () The timing of androgen ablation therapy and/or chemotherapy in the treatment of prostatic by: 3.

This chapter discusses clinical features, epidemiology, and management/treatment strategies for resistance to hormones such as thyroid receptor alpha and beta resistance to thyroid hormone (TR-alpha RTH and TR-beta RTH, respectively). It also discusses androgen insensitivity, as in resistance to testosterone and dihydrotestosterone, which can result in complete, partial, and Author: Helen E.

Turner. Hyperthyroidism (secondary to the homology between the beta-subunits of hCG and thyroid-stimulating hormone (TSH), which causes hCG to have weak TSH-like activity). Preeclampsia early in pregnancy. The most common antecedent pregnancy in Author: PDQ Adult Treatment Editorial Board.

provides accurate and independent information on more t prescription drugs, over-the-counter medicines and natural products. This material is provided for educational purposes only and is not intended for medical advice, diagnosis or treatment. Data sources include IBM Watson Micromedex (updated 4 May ), Cerner Multum™ (updated 4 May.

About Neoplasia, Estrogen Dependent: Neoplasia is a medical term describing an abnormal mass of tissue (tumor) caused by an abnormal group of rapidly dividing estrogen dependant neoplasia is a type of breast tumor that is dependent on estrogen for growth.

Stress creates hormonal responses that cause weight gain and insulin resistance. Cortisol is an adrenal hormone that helps you to run faster, see further, hear better and pump fuel into your bloodstream for quick energy.

It is the hormone that helps us survive in the face of true danger. It also shuts down digestion and slows your g: neoplasia. Title: Evidence for a Putative Relationship Between Type 2 Diabetes and Neoplasia with Particular Reference to Breast Cancer: Role of Hormones, Growth Factors and Specific Receptors VOLUME: 4 ISSUE: 1 Author(s):E.

Guastamacchia, F. Resta, V. Triggiani, A. Liso, B. Licchelli, S. Ghiyasaldin, C. Sabba and E. Tafaro Affiliation:Section of Endocrinology and Cited by: Common LHRH drugs include goserelin (Zoladex) and leuprolide (Lupron).

They can be used alone or with other hormone drugs (tamoxifen, aromatase inhibitors, fulvestrant) as hormone therapy in pre-menopausal women.

Chemotherapy drugs: Some chemo drugs can damage the ovaries of pre-menopausal women so they no longer make estrogen. Ovarian function. In Cancer Drug Resistance, leading scientists from the best academic institutions and industrial laboratories summarize and synthesize the latest discoveries concerning the changes that occur in tumor cells as they develop resistance to a wide variety of anticancer therapeutics, as well as suggest new approaches to the biology of drug Format: Hardcover.

Syndromes of Hormone Resistance on the Hypo-thalamic-Pituitary-Thyroid Axis Kluwer Academic Publishers Group, Dordrecht, Netherlands A book in the Endocrine Updates series.

Analogous patterns of IGF-1R overexpression in NSCLC drug resistance have been demonstrated with erlotinib, another EGFR-TKI. 55 Furthermore, in tumors that preferentially respond to erlotinib therapy due to an EGFR-activating mutation in e crosstalk between IGF-1R and epithelial-mesenchymal transition (EMT) signaling can rapidly confer drug Cited by: New Experimental Modalities in the Control of Neoplasia.

Editors: Chandra, Prakash (Ed.) such as the role of gene amplification in drug resistance and the app­ roaches to the reversal of resistance in drug refractory cancer, the role of membranes in designing useful strategies, the identification of new enzymic targets in some types of.

Get this from a library. DRUG AND HORMONE RESISTANCE IN NEOPLASIA: volume 2, Clinical concepts. [Nicholas Bruchovsky; James H Goldie;] -- Published in In Volume 2 the clinical aspects of drug and hormone resistance are discussed.

The Editors have concentrated on malignancies of the breast, endometrium, and prostate as relevant. Journal of Mammary Gland Biology and Neoplasia. All Volumes & Issues. Vol Issue 1, March Drug Resistance in Stem Cells and Breast Cancer. ISSN: (Print) (Online) Hormone-Induced Protection Against Breast Cancer.

Lakshmi Sivaraman, Daniel Medina Page Below is a list of common medications used to treat or reduce the symptoms of hormone+receptor+positive+breast+cancer. Follow the links to read common uses, side effects, dosage details and read.

Endocrine therapy has historically formed the basis of treatment of metastatic hormone receptor-positive breast cancer. The development of endocrine resistance has led to the development of newer endocrine drug combinations. Use of the CDK4/6 inhibitors has significantly improved progression-free survival in this group of patients.

There are multiple Cited by: 2. DES (diethylstilbestrol) is a man-made (synthetic) form of estrogen, a female hormone. Doctors prescribed it from until to help some pregnant women who had had miscarriages or premature deliveries. At that time it was believed that these problems might have been caused by low levels of estrogen in the woman’s body.Antibiotic-resistant bacteria sicken more than two million Americans each year and account for at le deaths.

The main cause? Overuse of antibiotics. A new report from the Centers for Disease Control and Prevention, Antibiotic Resistance Threats in the United States,details the health and financial costs of antibiotic resistance in the United States.

In terms of health.Yes. Most breast cancers are ER positive, and clinical trials have tested whether hormone therapy can be used to prevent breast cancer in women who are at increased risk of developing the disease. A large NCI-sponsored randomized clinical trial called the Breast Cancer Prevention Trial found that tamoxifen, taken for 5 years, reduced the risk of developing invasive breast .